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Irbesartan: Angiotensin II is a potent vasoconstrictor shaped from angiotensin I within a response catalyzed by angiotensin-converting enzyme (ACE, kininase II). Angiotensin II would be the principal pressor agent of the RAS and in addition stimulates aldosterone synthesis and secretion by adrenal cortex, cardiac contraction, renal resorption of sodium, exercise with the sympathetic anxious procedure, and clean muscle mobile progress.

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Hydrochlorothiazide: Hydrochlorothiazide is often a thiazide diuretic. Thiazides influence the renal tubular mechanisms of electrolyte reabsorption, directly expanding excretion of sodium and chloride in around equivalent quantities. Indirectly, the diuretic action of hydrochlorothiazide cuts down plasma volume, with consequent raises in plasma renin action, will increase in aldosterone secretion, improves in urinary potassium reduction, and decreases in serum potassium.

Angiotensin II would or else bind into the AT1 receptor, inducing vasoconstriction and aldosterone secretion, boosting blood pressure level.

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